^^^yup

When you gonna crack that bottle? You gonna up the test alongside the DMZ?

5-alpha-reductase is the enzyme that converts testosterone to DHT (dihydro-testosterone). DHT is a highly androgenic, less anabolic hormone compared to test (it's actually responsible for a lot of testosterone's perceived androgenicity), responsible for developing chest hair, deep voices (during puberty), and that "hard and dry" muscle look. Unfortunately, it's also responsible for balding.

Finasteride is a 5-AR inhibitor, it blocks the 5-alpha-reductase from working, stopping the conversion of test to DHT. It's very basically an anti-androgen, thus stopping baldness.

When you're taking deca (or any other nandrolone-containing steroid such as NPP), 5-AR will eventually convert the deca (or NPP) to DHN. DHN is actually extremely mild and much less androgenic than DHT. Deca itself isn't even a strong androgen to begin with.

Finasteride will just block DHN production and won't do anything to combat hair loss if you're on deca (deca and DHN are only mildly androgenic anyway). It will stop the deca from becoming DHN, leaving it to be converted to something estrogenic, or interact with progesterone receptors instead.

In summary - waste of pills, won't stop baldness (cause there isn't any), increases estrogen.

Dude thanks for the great explanation, honestly refreshing to have someone who obviously care about the science behind this. I didn't know that about nandrolone and DHN. Learn something new every day.

And yes, I know I mentioned it becomes estrogenic, even though it doesn't aromatize. It's true that nandrolone doesn't interact with aromatase, but there's actually another mechanism (that isn't well understood yet) by which it becomes estrogenic. Most problems would come from progesterone first though (it is a 19-nor after all), then estrogen.

I'm gonna go a little in depth, so we won't have to answer again if a newbie asks for more details. You'll know most of this, so bear with me:

5-alpha-reductase is the enzyme that converts testosterone to DHT (dihydro-testosterone). DHT is a highly androgenic, less anabolic hormone compared to test (it's actually responsible for a lot of testosterone's perceived androgenicity), responsible for developing chest hair, deep voices (during puberty), and that "hard and dry" muscle look. Unfortunately, it's also responsible for balding.

Finasteride is a 5-AR inhibitor, it blocks the 5-alpha-reductase from working, stopping the conversion of test to DHT. It's very basically an anti-androgen, thus stopping baldness.

When you're taking deca (or any other nandrolone-containing steroid such as NPP), 5-AR will eventually convert the deca (or NPP) to DHN. DHN is actually extremely mild and much less androgenic than DHT. Deca itself isn't even a strong androgen to begin with.

Finasteride will just block DHN production and won't do anything to combat hair loss if you're on deca (deca and DHN are only mildly androgenic anyway). It will stop the deca from becoming DHN, leaving it to be converted to something estrogenic, or interact with progesterone receptors instead.

In summary - waste of pills, won't stop baldness (cause there isn't any), increases estrogen.

Why no finasteride with it??

correct. i pin npp every other day. and test is a must.